G. Twig

Molecular Biology · 2008–2024

A researcher in the Science corpus: 6 papers; trusts 8 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

Whom G. Twig trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Guido Kroemer
  2. Hsiuchen Chen
  3. D. Klionsky
  4. Naotada Ishihara
  5. D. C. Chan
  6. S. Cipolat
  7. R. Youle
  8. Derek P. Narendra
  9. Zhiyin Song
  10. Koji Okamoto

Who trusts G. Twig overall

  1. R. Youle
  2. B. Westermann
  3. W. Ding
  4. S. Ghavami
  5. Alicia M. Pickrell
  6. Ji Zhang
  7. Derek P. Narendra
  8. M. Liesa
  9. A. M. van der Bliek
  10. Ghazaleh Ashrafi

Papers from G. Twig's perspective

Ordered by how strongly the researchers G. Twig trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Disruption of Fusion Results in Mitochondrial Heterogeneity and Dysfunction* (Journal of Biological Chemistry)
  2. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  3. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
  4. Roles of the mammalian mitochondrial fission and fusion mediators Fis1, Drp1, and Opa1 in apoptosis. (Molecular Biology of the Cell)
  5. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
  6. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  7. OPA1 requires mitofusin 1 to promote mitochondrial fusion. (Proceedings of the National Academy of Sciences of the United States of America)
  8. The Mitochondrial Protein hFis1 Regulates Mitochondrial Fission in Mammalian Cells through an Interaction with the Dynamin-Like Protein DLP1 (Molecular and Cellular Biology)
  9. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  10. Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)

Their papers