Alicia M. Pickrell

Epidemiology · 2015–2021

A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 2 researchers here, as inferred from the citation graph.

Subfields

• Epidemiology — 35 citations into it; #969 of 2,148 by trust here, #1,104 by citations
• Molecular Biology — 34 citations into it; #10,169 of 17,773 by trust here, #10,193 by citations
• Neurology — 30 citations into it; #661 of 1,455 by trust here, #675 by citations
• Physiology — 6 citations into it

Whom Alicia M. Pickrell trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. R. Youle
2. Derek P. Narendra
3. G. Twig
4. S. Jin
5. M. Lazarou
6. Koji Yamano
7. H. Ko
8. Hsiuchen Chen

Who trusts Alicia M. Pickrell overall

1. S. Camandola
2. Mark P. Mattson
3. Lauren E Tebay
4. H. Robertson
5. T. Nguyen
6. Wen Chen

Papers from Alicia M. Pickrell's perspective

Ordered by how strongly the researchers Alicia M. Pickrell trusts endorse each paper. Same corpus, a different order for every researcher.

1. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
2. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
3. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
4. Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)
5. Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase (Nature Genetics)
6. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
7. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)
8. The PINK1/Parkin pathway regulates mitochondrial morphology (Proceedings of the National Academy of Sciences of the United States of America)
9. Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress (Proceedings of the National Academy of Sciences of the United States of America)
10. Hereditary Early-Onset Parkinson's Disease Caused by Mutations in PINK1 (Science)

Their papers

• Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1 (2021) — 3,841 citations
• The Roles of PINK1, Parkin and Mitochondrial Fidelity in Parkinson's Disease (2015) — 2,096 citations