Derek P. Narendra
Epidemiology · 2008–2019
A researcher in the Science corpus: 9 papers; trusts 6 researchers and is trusted by 53 researchers here, as inferred from the citation graph.
Subfields
- Epidemiology — 204 citations into it; #23 of 2,148 by trust here, #65 by citations
- Molecular Biology — 157 citations into it; #1,744 of 17,773 by trust here, #2,259 by citations
- Cell Biology — 89 citations into it
- Neurology — 76 citations into it; #37 of 1,455 by trust here, #25 by citations
- Cellular and Molecular Neuroscience — 39 citations into it
- Geriatrics and Gerontology — 5 citations into it
Whom Derek P. Narendra trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- V. Kirkin
- D. Suen
- Ivan Dikic
- G. Twig
- R. Youle
- Hsiuchen Chen
- A. Schapira
- Naotada Ishihara
- S. Jin
- Douglas C. Wallace
Who trusts Derek P. Narendra overall
- R. Youle
- S. Jin
- Y. Wong
- Nicole Exner
- Gian-Luca McLelland
- N. Mizushima
- W. Ding
- M. Lazarou
- A. Ordureau
- Douglas R. Green
Papers from Derek P. Narendra's perspective
Ordered by how strongly the researchers Derek P. Narendra trusts endorse each paper. Same corpus, a different order for every researcher.
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
- Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- Disruption of Fusion Results in Mitochondrial Heterogeneity and Dysfunction* (Journal of Biological Chemistry)
- The mitochondrial permeability transition initiates autophagy in rat hepatocytes (The FASEB Journal)
- Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. (Proceedings of the National Academy of Sciences of the United States of America)
- Selective degradation of mitochondria by mitophagy. (Archives of Biochemistry and Biophysics)
- PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (2008) — 4,017 citations
- p62/SQSTM1 is required for Parkin-induced mitochondrial clustering but not mitophagy; VDAC1 is dispensable for both (2010) — 782 citations
- Proteasome and p97 mediate mitophagy and degradation of mitofusins induced by Parkin (2010) — 1,356 citations
- Parkin and PINK1 mitigate STING-induced inflammation (2018) — 1,155 citations
- Identification of novel risk loci, causal insights, and heritable risk for Parkinson’s disease: a meta-genome wide association study (2019) — 1,651 citations
- Guidelines for the use and interpretation of assays for monitoring autophagy (2012) — 4,856 citations
- Mechanisms of mitophagy (2011) — 3,203 citations
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (2010) — 2,915 citations
- Mitochondrial membrane potential regulates PINK1 import and proteolytic destabilization by PARL (2010) — 1,343 citations