Vincent Soubannier

Epidemiology · 2002–2014

A researcher in the Science corpus: 3 papers; trusts 6 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

• Epidemiology — 26 citations into it; #1,675 of 2,148 by trust here, #1,182 by citations
• Neurology — 19 citations into it; #1,125 of 1,455 by trust here, #1,146 by citations
• Molecular Biology — 18 citations into it; #15,069 of 17,773 by trust here, #8,905 by citations
• Physiology — 9 citations into it

Whom Vincent Soubannier trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Derek P. Narendra
2. N. Mizushima
3. M. V. Vander Heiden
4. D. Klionsky
5. R. Youle
6. Beth Levine
7. Zhiping Xie
8. D. Suen
9. Lewis C. Cantley
10. M. Lazarou

Who trusts Vincent Soubannier overall

1. Douglas R. Green
2. I. Slaninová
3. Ayumu Sugiura
4. Gian-Luca McLelland
5. Beth Levine

Papers from Vincent Soubannier's perspective

Ordered by how strongly the researchers Vincent Soubannier trusts endorse each paper. Same corpus, a different order for every researcher.

1. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
2. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
3. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
4. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
5. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
6. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
7. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
8. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
9. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)
10. Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)

Their papers

• The ATP synthase is involved in generating mitochondrial cristae morphology (2002) — 794 citations
• Parkin and PINK1 function in a vesicular trafficking pathway regulating mitochondrial quality control (2014) — 722 citations
• A vesicular transport pathway shuttles cargo from mitochondria to lysosomes. (2012) — 720 citations