Gian-Luca McLelland

Epidemiology · 2012–2014

A researcher in the Science corpus: 3 papers; trusts 10 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

• Epidemiology — 45 citations into it; #1,674 of 2,148 by trust here, #925 by citations
• Neurology — 43 citations into it; #1,128 of 1,455 by trust here, #1,146 by citations
• Molecular Biology — 39 citations into it; #15,070 of 17,773 by trust here, #9,030 by citations
• Physiology — 19 citations into it

Whom Gian-Luca McLelland trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Derek P. Narendra
2. R. Youle
3. N. Mizushima
4. Zhiping Xie
5. M. Lazarou
6. D. Klionsky
7. S. Jin
8. D. Suen
9. Vincent Soubannier
10. Beth Levine

Who trusts Gian-Luca McLelland overall

1. I. Slaninová
2. Ayumu Sugiura
3. Prashant Mishra
4. S. Weinberg
5. Douglas R. Green

Papers from Gian-Luca McLelland's perspective

Ordered by how strongly the researchers Gian-Luca McLelland trusts endorse each paper. Same corpus, a different order for every researcher.

1. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
2. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
3. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
4. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
5. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
6. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
7. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
8. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
9. Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)
10. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)

Their papers

• Parkin and PINK1 function in a vesicular trafficking pathway regulating mitochondrial quality control (2014) — 722 citations
• A vesicular transport pathway shuttles cargo from mitochondria to lysosomes. (2012) — 720 citations
• A new pathway for mitochondrial quality control: mitochondrial‐derived vesicles (2014) — 791 citations