M. Lazarou

Epidemiology · 2010–2018

A researcher in the Science corpus: 6 papers; trusts 6 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

Whom M. Lazarou trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Derek P. Narendra
  2. S. Jin
  3. R. Youle
  4. D. Suen

Who trusts M. Lazarou overall

  1. Nicole Exner
  2. T. Nguyen
  3. Gian-Luca McLelland
  4. Gabriele Zaffagnini
  5. A. Ordureau
  6. R. Youle
  7. A. Lutz
  8. Mashun Onishi
  9. B. Padman
  10. M. Fricker

Papers from M. Lazarou's perspective

Ordered by how strongly the researchers M. Lazarou trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  2. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
  3. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  4. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  5. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
  6. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
  7. Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. (Proceedings of the National Academy of Sciences of the United States of America)
  8. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
  9. Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase (Nature Genetics)
  10. Mitochondrial Dysfunction and Oxidative Damage in parkin-deficient Mice* (Journal of Biological Chemistry)

Their papers