Mitochondria: In Sickness and in Health
By J. Nunnari, A. Suomalainen. Cell, 2012
3,213 citations recorded by the source corpus.
Subfields: Clinical Biochemistry, Epidemiology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/e49212cac39325f24f7b22160861f0ed73b0f975
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Clinical Biochemistry
In Epidemiology
In Molecular Biology
- D. Finley
- Víctor M. Víctor
- Robert W. Williams
- Yan Pang
- Xianqun Fan
- Xieyang Xu
- Steven P. Gygi
- Johan Auwerx
Citations in the corpus
- Multi-omics analysis identifies ATF4 as a key regulator of the mitochondrial stress response in mammals (2017, background): “Mitochondria are key for energy metabolism within the cell, as they produce most of the cellular ATP through oxidative phosphorylation (OXP HOS) and are essential for regulating intermediate metabolism (Nunnari and Suomalainen, 2012).”
- Role of ROS and RNS Sources in Physiological and Pathological Conditions (2016, background): “On the other hand, mitochondrial division should create organelles of the appropriate size for transport along actin or microtubule networks [125].”
- Mitochondrial ROS signaling in organismal homeostasis. (2015, background): “With regard to genetics, the importance of these pathways is underscored by the fact that inherited mitochondrial diseases are caused by mutations in genes encoding proteins involved in each of these processes that can be inheritedmaternally (mtDNA mutations) or in a Mendelian fashion (nuclear gene ”
- Mitochondria in oxidative stress, inflammation and aging: from mechanisms to therapeutic advances (2025, positive): “24 As mitochondria are responsible for energy production and cellular respiration, their dysfunction will result in muscle diseases, such as mitochondrial myopathies and myotonic dystrophy.”
- Mechanisms of mitochondrial fission and fusion. (2013, background): “are caused by mutations in fission and fusion proteins and the discovery of numerous connections with apoptosis and mitophagy (Westermann 2010; Chan 2012; Nunnari and Suomalainen 2012; Youle and van der Bliek 2012).”