Nicole Exner
Neurology · 2012
A researcher in the Science corpus: 1 paper; trusts 7 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Neurology — 73 citations into it
- Molecular Biology — 49 citations into it
- Epidemiology — 24 citations into it
- Physiology — 23 citations into it
Whom Nicole Exner trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Derek P. Narendra
- S. Jin
- M. Lazarou
- Kelvin C. Luk
- M. Farrer
- L. A. Kane
- R. Youle
- M. Dodson
- L. Volpicelli-Daley
- S. Gandhi
Papers from Nicole Exner's perspective
Ordered by how strongly the researchers Nicole Exner trusts endorse each paper. Same corpus, a different order for every researcher.
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
- Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase (Nature Genetics)
- Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)
- Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress (Proceedings of the National Academy of Sciences of the United States of America)
- Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
- Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)