S. Korsmeyer
Molecular Biology · 2001–2012
A researcher in the Science corpus: 16 papers; trusts 1 researcher and is trusted by 18 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 87 citations into it; #2,130 of 17,773 by trust here, #662 by citations
Whom S. Korsmeyer trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts S. Korsmeyer overall
Papers from S. Korsmeyer's perspective
Ordered by how strongly the researchers S. Korsmeyer trusts endorse each paper. Same corpus, a different order for every researcher.
- BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak. (Genes & Development)
- Bid Induces the Oligomerization and Insertion of Bax into the Outer Mitochondrial Membrane (Molecular and Cellular Biology)
- PUMA, a novel proapoptotic gene, is induced by p53. (Molecules and Cells)
- Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function. (Molecules and Cells)
- Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation. (Genes & Development)
- Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. (Cancer Cell)
- Conjugation of arginine oligomers to cyclosporin A facilitates topical delivery and inhibition of inflammation (Nature Medicine)
- An inhibitor of Bcl-2 family proteins induces regression of solid tumours (Nature)
- Activation of Apoptosis in Vivo by a Hydrocarbon-Stapled BH3 Helix (Science)
- A critical role for Dnmt1 and DNA methylation in T cell development, function, and survival. (Immunity)
Their papers
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (2001) — 4,220 citations
- A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis. (2002) — 1,057 citations
- Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. (2002) — 1,714 citations
- BAX and BAK Regulation of Endoplasmic Reticulum Ca2+: A Control Point for Apoptosis (2003) — 1,488 citations
- An inhibitor of Bcl-2 family proteins induces regression of solid tumours (2005) — 3,422 citations
- VDAC2 Inhibits BAK Activation and Mitochondrial Apoptosis (2003) — 854 citations
- Mitochondria primed by death signals determine cellular addiction to antiapoptotic BCL-2 family members. (2006) — 1,299 citations
- Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia. (2005) — 838 citations
- Mechanisms of cytochrome c release by proapoptotic BCL-2 family members. (2003) — 734 citations
- Exercise–induced BCL2–regulated autophagy is required for muscle glucose homeostasis (2012) — 1,102 citations
- Obligate Role of Anti-Apoptotic MCL-1 in the Survival of Hematopoietic Stem Cells (2005) — 613 citations
- Activation of Apoptosis in Vivo by a Hydrocarbon-Stapled BH3 Helix (2004) — 1,269 citations
- Development and maintenance of B and T lymphocytes requires antiapoptotic MCL-1 (2003) — 855 citations
- BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis (2003) — 740 citations
- MLL translocations specify a distinct gene expression profile that distinguishes a unique leukemia (2002) — 1,548 citations
- Bcl2 regulation by the melanocyte master regulator Mitf modulates lineage survival and melanoma cell viability. (2002) — 779 citations