A. Letai

Molecular Biology · 2002–2020

A researcher in the Science corpus: 16 papers; trusts 18 researchers and is trusted by 11 researchers here, as inferred from the citation graph.

Subfields

Whom A. Letai trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. X. Wang
  2. W. Zong
  3. Simon N. Willis
  4. Lin Chen
  5. T. Kuwana
  6. K. Nakano
  7. J. Chipuk
  8. M. Castedo
  9. A. Strasser
  10. S. Korsmeyer

Who trusts A. Letai overall

  1. J. Chipuk
  2. Douglas R. Green
  3. Jerry M. Adams
  4. Thomas G. Cotter
  5. Simone Fulda
  6. A. Strasser
  7. Simon N. Willis
  8. J. Brunelle
  9. M. Fricker
  10. Suzanne Cory

Papers from A. Letai's perspective

Ordered by how strongly the researchers A. Letai trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis. (Science)
  2. BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak. (Genes & Development)
  3. PUMA, a novel proapoptotic gene, is induced by p53. (Molecules and Cells)
  4. BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis. (Molecules and Cells)
  5. Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
  6. Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function. (Molecules and Cells)
  7. Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation. (Genes & Development)
  8. The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. (Molecules and Cells)
  9. An inhibitor of Bcl-2 family proteins induces regression of solid tumours (Nature)
  10. The expanding role of mitochondria in apoptosis. (Genes & Development)

Their papers