J. Brunelle

Molecular Biology · 2005–2009

A researcher in the Science corpus: 2 papers; trusts 3 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom J. Brunelle trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. W. Pfaffl
  2. X. Wang
  3. A. Letai
  4. K. Nakano
  5. T. Kuwana
  6. S. Korsmeyer
  7. G. Semenza

Who trusts J. Brunelle overall

  1. Jung-whan Kim
  2. R. Guzy
  3. G. Semenza
  4. R. Fukuda
  5. Kyle D Mansfield
  6. C. V. Dang
  7. Toren Finkel
  8. M. S. Ola

Papers from J. Brunelle's perspective

Ordered by how strongly the researchers J. Brunelle trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis. (Science)
  2. PUMA, a novel proapoptotic gene, is induced by p53. (Molecules and Cells)
  3. Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. (Cancer Cell)
  4. Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function. (Molecules and Cells)
  5. Mitochondria primed by death signals determine cellular addiction to antiapoptotic BCL-2 family members. (Cancer Cell)
  6. Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
  7. An inhibitor of Bcl-2 family proteins induces regression of solid tumours (Nature)
  8. The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. (Molecules and Cells)
  9. The expanding role of mitochondria in apoptosis. (Genes & Development)
  10. BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly. (Molecules and Cells)

Their papers