K. Boniface
Nature Immunology · 2005–2007
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Whom K. Boniface trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts K. Boniface overall
Papers from K. Boniface's perspective
Ordered by how strongly the researchers K. Boniface trusts endorse each paper. Same corpus, a different order for every researcher.
- IL-22 increases the innate immunity of tissues. (Immunity)
- Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12.
- Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
- Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
- A human interleukin-12/23 monoclonal antibody for the treatment of psoriasis. (New England Journal of Medicine)
- TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
- IL‐22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasis (European Journal of Immunology)
- A large-scale genetic association study confirms IL12B and leads to the identification of IL23R as psoriasis-risk genes. (American Journal of Human Genetics)
- Interleukin-10 and the interleukin-10 receptor. (Annual Review of Immunology)