Christopher M. Overall

Molecular Biology · 2000–2011

A researcher in the Science corpus: 8 papers; trusts 14 researchers and is trusted by 17 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 58 citations into it; #3,487 of 17,773 by trust here, #6,028 by citations
• Cancer Research — 49 citations into it; #211 of 1,780 by trust here, #338 by citations
• Oncology — 43 citations into it; #133 of 1,698 by trust here, #362 by citations
• Immunology and Allergy — 19 citations into it; #53 of 206 by trust here, #25 by citations
• Hematology — 16 citations into it
• Immunology — 12 citations into it
• Biomedical Engineering — 6 citations into it

Whom Christopher M. Overall trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. C. López‐Otín
2. M. Egeblad
3. Lisa M. Coussens
4. G. A. McQuibban
5. Richard Bonneau
6. Zena Werb
7. Ralph Weissleder
8. Eric J. Topol
9. L. Matrisian
10. J. Gong

Who trusts Christopher M. Overall overall

1. B. Turk
2. C. López‐Otín
3. Erik Sahai
4. Francis G. Spinale
5. J. Vandooren
6. Andrea Page-McCaw
7. C. Malemud
8. P. Steeg
9. Chunhong Yan
10. G. Opdenakker

Papers from Christopher M. Overall's perspective

Ordered by how strongly the researchers Christopher M. Overall trusts endorse each paper. Same corpus, a different order for every researcher.

1. New functions for the matrix metalloproteinases in cancer progression (Nature Reviews. Cancer)
2. Matrix metalloproteinase-9 triggers the angiogenic switch during carcinogenesis (Nature Cell Biology)
3. Development of matrix metalloproteinase inhibitors in cancer therapy. (Journal of the National Cancer Institute)
4. TIMP-3 Is a Potent Inhibitor of Aggrecanase 1 (ADAM-TS4) and Aggrecanase 2 (ADAM-TS5)*
5. Neutrophil gelatinase B potentiates interleukin-8 tenfold by aminoterminal processing, whereas it degrades CTAP-III, PF-4, and GRO-alpha and leaves RANTES and MCP-2 intact. (Blood)
6. Impaired endochondral ossification and angiogenesis in mice deficient in membrane-type matrix metalloproteinase I. (Proceedings of the National Academy of Sciences of the United States of America)
7. How matrix metalloproteinases regulate cell behavior. (Annual Review of Cell and Developmental Biology)
8. Printing proteins as microarrays for high-throughput function determination. (Science)
9. Matrix metalloproteinases in tumor invasion and metastasis. (Seminars in Cancer Biology)
10. Membrane type I matrix metalloproteinase usurps tumor growth control imposed by the three-dimensional extracellular matrix. (Cell)

Their papers

• Protease degradomics: A new challenge for proteomics (2002) — 769 citations
• Strategies for MMP inhibition in cancer: innovations for the post-trial era (2002) — 1,357 citations
• Validating matrix metalloproteinases as drug targets and anti-targets for cancer therapy (2006) — 1,111 citations
• Targeting tumor hypoxia: suppression of breast tumor growth and metastasis by novel carbonic anhydrase IX inhibitors. (2011) — 675 citations
• Multi-step pericellular proteolysis controls the transition from individual to collective cancer cell invasion (2007) — 1,033 citations
• Human and mouse proteases: a comparative genomic approach (2003) — 944 citations
• Matrix metalloproteinase processing of monocyte chemoattractant proteins generates CC chemokine receptor antagonists with anti-inflammatory properties in vivo. (2002) — 649 citations
• Inflammation dampened by gelatinase A cleavage of monocyte chemoattractant protein-3. (2000) — 822 citations