Francis G. Spinale

Cancer Research · 2002–2011

A researcher in the Science corpus: 3 papers; trusts 14 researchers and is trusted by 13 researchers here, as inferred from the citation graph.

Subfields

Whom Francis G. Spinale trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. Zile
  2. B. Jugdutt
  3. Christopher M. Overall
  4. D. Brutsaert
  5. Matthew P. Vincenti
  6. Véronique Baud
  7. Chunhong Yan
  8. R. Visse
  9. K. Brew
  10. Hideaki Nagase

Who trusts Francis G. Spinale overall

  1. Nikolaos G. Frangogiannis
  2. W. Qin
  3. Yide Cao
  4. K. Porter
  5. A. Takawale
  6. Liangpeng Li
  7. Scott C. Weaver
  8. M. Drazner
  9. R. Visse
  10. Wen Chen

Papers from Francis G. Spinale's perspective

Ordered by how strongly the researchers Francis G. Spinale trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Strategies for MMP inhibition in cancer: innovations for the post-trial era (Nature Reviews. Cancer)
  2. Tissue inhibitors of metalloproteinases: evolution, structure and function. (Biochimica et Biophysica Acta)
  3. S-Nitrosylation of Matrix Metalloproteinases: Signaling Pathway to Neuronal Cell Death (Science)
  4. The cysteine switch: a principle of regulation of metalloproteinase activity with potential applicability to the entire matrix metalloproteinase gene family.
  5. Diastolic heart failure--abnormalities in active relaxation and passive stiffness of the left ventricle. (New England Journal of Medicine)
  6. Structure and function of matrix metalloproteinases and TIMPs. (Cardiovascular Research)
  7. Signal transduction by tumor necrosis factor and its relatives. (Trends in Cell Biology)
  8. Regulation of matrix metalloproteinases: An overview (Molecular and Cellular Biochemistry)
  9. Transcriptional regulation of collagenase (MMP-1, MMP-13) genes in arthritis: integration of complex signaling pathways for the recruitment of gene-specific transcription factors (Arthritis Research & Therapy)
  10. Prediction of the risk of myocardial infarction from polymorphisms in candidate genes. (New England Journal of Medicine)

Their papers