Thomas Brabletz

Molecular Biology · 2001–2021

A researcher in the Science corpus: 15 papers; trusts 41 researchers and is trusted by 18 researchers here, as inferred from the citation graph.

Subfields

Whom Thomas Brabletz trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. J. P. Thiery
  2. R. Weinberg
  3. Anurag Singh
  4. M. Nieto
  5. Raghu Kalluri
  6. H. Peinado
  7. A. Cano
  8. Christine L. Chaffer
  9. Sendurai A Mani
  10. Jing Yang

Who trusts Thomas Brabletz overall

  1. M. Nieto
  2. R. Weinberg
  3. D. Hanahan
  4. Simone Brabletz
  5. J. Medema
  6. J. P. Thiery
  7. David M. Gonzalez
  8. V. Mittal
  9. O. Schmalhofer
  10. Raghu Kalluri

Papers from Thomas Brabletz's perspective

Ordered by how strongly the researchers Thomas Brabletz trusts endorse each paper. Same corpus, a different order for every researcher.

  1. The epithelial-mesenchymal transition generates cells with properties of stem cells. (Cell)
  2. Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis. (Cell)
  3. Migrating cancer stem cells — an integrated concept of malignant tumour progression (Nature Reviews. Cancer)
  4. Prospective identification of tumorigenic breast cancer cells (Proceedings of the National Academy of Sciences of the United States of America)
  5. The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2. (Genes & Development)
  6. Complex networks orchestrate epithelial–mesenchymal transitions (Nature reviews. Molecular cell biology)
  7. Epithelial–mesenchymal transitions in tumour progression (Nature Reviews. Cancer)
  8. The two-handed E box binding zinc finger protein SIP1 downregulates E-cadherin and induces invasion. (Molecules and Cells)
  9. The transcription factor Snail is a repressor of E-cadherin gene expression in epithelial tumour cells (Nature Cell Biology)
  10. DeltaEF1 is a transcriptional repressor of E-cadherin and regulates epithelial plasticity in breast cancer cells (Oncogene)

Their papers