E-cadherin, β-catenin, and ZEB1 in malignant progression of cancer

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Hallmarks of cancer: the next generation. (2011, positive): “…tumor types and have been shown in experimental models of carcinoma formation to be causally important for programming invasion; some have been found to elicit metastasis when ectopically overexpressed (Micalizzi et al., 2010; Taube et al., 2010; Schmalhofer et al., 2009; Yang and Weinberg, 2008).”
Fap2 Mediates Fusobacterium nucleatum Colorectal Adenocarcinoma Enrichment by Binding to Tumor-Expressed Gal-GalNAc (2016, background): “A uthor M anuscript A uthor M anuscript A uthor M anuscript A uthor M anuscript on many cell types (Frixen et al., 1991; Heng and Painter, 2008), and its expression levels and cellular localization patterns in dysplasia and neoplasia can vary (Jiang et al., 2015; Schmalhofer et al., 2009).”
E-cadherin–integrin crosstalk in cancer invasion and metastasis (2013, background): “…axis can lead to the nuclear translocation of b-catenin following disruption of AJs, and the subsequent activation of its transcriptional activity (Coluccia et al., 2006; Koenig et al., 2006) that is commonly associated with more invasive and aggressive tumours (Schmalhofer et al., 2009).”