F. Shao

Molecular Biology · 2003–2021

A researcher in the Science corpus: 17 papers; trusts 6 researchers and is trusted by 12 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 348 citations into it; #4,144 of 17,773 by trust here, #947 by citations
• Immunology — 210 citations into it; #611 of 2,160 by trust here, #210 by citations

Whom F. Shao trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. B. Cookson
2. Jianjin Shi
3. M. Torres

Who trusts F. Shao overall

1. Jonathan D. G. Jones
2. Junying Yuan
3. Jane Glazebrook
4. Alok Kumar Pandey

Papers from F. Shao's perspective

Ordered by how strongly the researchers F. Shao trusts endorse each paper. Same corpus, a different order for every researcher.

1. Caspase‐1‐dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages (Cellular Microbiology)
2. Non-canonical inflammasome activation targets caspase-11 (Nature)
3. Salmonella induces macrophage death by caspase‐1‐dependent necrosis (Molecular Microbiology)
4. Caspase-1-induced pyroptosis is an innate immune effector mechanism against intracellular bacteria (Nature Immunology)
5. The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta. (Molecules and Cells)
6. The NLRC4 inflammasome receptors for bacterial flagellin and type III secretion apparatus (Nature)
7. Inflammasome - activated gasdermin D causes pyroptosis by forming membrane pores (Nature)
8. Cleavage of DFNA5 by caspase-3 during apoptosis mediates progression to secondary necrotic/pyroptotic cell death (Nature Communications)
9. Pyroptotic cell death defends against intracellular pathogens (Immunological Reviews)
10. Are innate immune signaling pathways in plants and animals conserved? (Nature Immunology)

Their papers

• Inflammatory caspases are innate immune receptors for intracellular LPS (2014) — 2,123 citations
• Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death (2015) — 5,789 citations
• Pyroptosis: Gasdermin-Mediated Programmed Necrotic Cell Death. (2017) — 2,670 citations
• Chemotherapy drugs induce pyroptosis through caspase-3 cleavage of a gasdermin (2017) — 2,745 citations
• Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 (2018) — 5,732 citations
• Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1 (2021) — 3,841 citations
• The NLRC4 inflammasome receptors for bacterial flagellin and type III secretion apparatus (2011) — 1,238 citations
• Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3–mediated pathway (2011) — 701 citations
• Pore-forming activity and structural autoinhibition of the gasdermin family (2016) — 1,946 citations
• Caspase-8 induces cleavage of gasdermin D to elicit pyroptosis during Yersinia infection (2018) — 794 citations
• Innate immune sensing of bacterial modifications of Rho GTPases by the Pyrin inflammasome (2014) — 771 citations
• Granzyme A from cytotoxic lymphocytes cleaves GSDMB to trigger pyroptosis in target cells (2020) — 1,099 citations
• The gasdermins, a protein family executing cell death and inflammation (2019) — 1,405 citations
• A bioorthogonal system reveals antitumour immune function of pyroptosis (2020) — 875 citations
• Structural Mechanism for GSDMD Targeting by Autoprocessed Caspases in Pyroptosis. (2020) — 645 citations
• A Pseudomonas syringae effector inactivates MAPKs to suppress PAMP-induced immunity in plants. (2007) — 689 citations
• Cleavage of Arabidopsis PBS1 by a Bacterial Type III Effector (2003) — 597 citations