G. Aghajanian
Cellular and Molecular Neuroscience · 2010–2016
A researcher in the Science corpus: 4 papers; trusts 2 researchers and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Cellular and Molecular Neuroscience — 50 citations into it; #2,255 of 2,998 by trust here, #821 by citations
- Molecular Biology — 15 citations into it
- Genetics — 8 citations into it
Whom G. Aghajanian trusts
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts G. Aghajanian
- G. Sanacora (in Cellular and Molecular Neuroscience)
- R. Duman (in Cellular and Molecular Neuroscience)
Papers from G. Aghajanian's perspective
Ordered by how strongly the researchers G. Aghajanian trusts endorse each paper. Same corpus, a different order for every researcher.
- VEGF is an essential mediator of the neurogenic and behavioral actions of antidepressants
- Hippocampal volume and depression: a meta-analysis of MRI studies. (American Journal of Psychiatry)
- Hypothalamic mTOR Signaling Regulates Food Intake (Science)
- Gliogenesis and glial pathology in depression. (CNS and Neurological Disorders - Drug Targets)
- Neuroimaging and neuropathological studies of depression: implications for the cognitive-emotional features of mood disorders. (Current Opinion in Neurobiology)
- Untreated depression and hippocampal volume loss. (American Journal of Psychiatry)
- Sustained hippocampal chromatin regulation in a mouse model of depression and antidepressant action (Nature Neuroscience)
- The yin and yang of neurotrophin action (Nature Reviews Neuroscience)
- mTOR-dependent synapse formation underlies the rapid antidepressant effects of NMDA antagonists (Science)
- Requirement of Hippocampal Neurogenesis for the Behavioral Effects of Antidepressants (Science)
Their papers
- Synaptic plasticity and depression: New insights from stress and rapid-acting antidepressants (2016) — 1,559 citations
- Synaptic Dysfunction in Depression: Potential Therapeutic Targets (2012) — 1,531 citations
- Glutamate NMDA receptor antagonists rapidly reverse behavioral and synaptic deficits caused by chronic stress exposure (2011) — 1,040 citations
- mTOR-dependent synapse formation underlies the rapid antidepressant effects of NMDA antagonists (2010) — 2,783 citations