Neuroimaging and neuropathological studies of depression: implications for the cognitive-emotional features of mood disorders.

By Wayne C. Drevets. Current Opinion in Neurobiology, 2001

1,172 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Pharmacology

View Paper: https://www.semanticscholar.org/paper/5dae67fe2d9034b26c9d49bfc81e3c82ee73614c

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Citations in the corpus

Neurocircuitry of Mood Disorders (2010, background): “For example, in the amygdala, resting metabolism was elevated in subjects classified as BD-depressed, familial pure depressive disease (FPDD), melancholic subtype, or responsive to a night of total sleep deprivation (reviewed in Drevets, 2001; Drevets et al, 2004a).”
Modulation of emotion by cognition and cognition by emotion (2007, background): “Current work using the current and related tasks is exploring these possibilities with patients with Generalized Anxiety Disorder and patients with Post Traumatic Stress Disorder.”
Automatic emotion regulation (2005, background): “…Mayberg and colleagues have proposed that sustained suppression of the SCC is necessary for full recovery from unipolar depression (Davidson, 2002; Drevets, 2001; Gotlib et al., 2005; Liotti et al., 2002; Mayberg, 2003; Shin, Kosslyn, McNally, & Alpert, 1997), and fear extinction in healthy…”
Depression: a case of neuronal life and death? (2004, background): “anji H, Drevets WC, Charney DS (2001): The cellular neurobiology of depression.”
A meta-analysis of emotional reactivity in major depressive disorder. (2008, background): “Using fMRI, a blunted response in the amygdala to facial expressions of fear (relative to neutral) has been observed in both depressed adults (Drevets, 2001) and depressed children (Thomas et al., 2001), which might be interpreted in terms of ECI.”
Efficient, Distributed and Interactive Neuroimaging Data Analysis Using the LONI Pipeline (2009, neutral): “…information from large cross-sectional or longitudinal studies in multiple sclerosis (Liu et al., 2005), Alzheimer’s disease (Fleisher et al., 2005), autism (Langen et al., 2007), depression (Drevets, 2001), schizophrenia (Narr et al., 2007), and studies of normal populations (Gogtay et al., 2006).”
Association between heart rate variability and fluctuations in resting-state functional connectivity (2012, background): “…has been linked to the sympathetic skin response during fear (Asahina et al., 2003; Williams et al., 2001), and shows hyperactivity in depression (Drevets, 2001), a disease having a characteristic decrease in HRV. Studies that have directly examined correlations between heart rate and amygdala…”
Hippocampal neurogenesis: Opposing effects of stress and antidepressant treatment (2006, background): “Fourth, the hippocampus shares anatomical connections with the amygdala and prefrontal cortex, two highly studied brain areas that have been implicated in mood and cognition (Drevets, 2001).”
Discovering Endophenotypes for Major Depression (2004, background): “Volume reductions in the ACC located ventrally (‘subgenual’) and anterior (‘pregenual’) to the genu of the corpus callosum have been implicated by numerous studies of mood disorders (Drevets, 2001).”
The molecular neurobiology of depression (2008, neutral): “Second, most depression occurs idiopathically, and the limited understanding of its aetiology is reflected as a list of risk factors, such as stressful life events, endocrine abnormalities (hypothyroidism and hypercortisolism), cancers (such as pancreatic adenocarcinoma and breast tumours) and side ”