Thomas A. Prolla
Molecular Biology · 2000–2013
A researcher in the Science corpus: 10 papers; trusts 4 researchers and is trusted by 4 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 65 citations into it; #16,257 of 17,773 by trust here, #3,514 by citations
- Geriatrics and Gerontology — 59 citations into it
- Statistics and Probability — 6 citations into it
Whom Thomas A. Prolla trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Thomas A. Prolla overall
Papers from Thomas A. Prolla's perspective
Ordered by how strongly the researchers Thomas A. Prolla trusts endorse each paper. Same corpus, a different order for every researcher.
- Premature ageing in mice expressing defective mitochondrial DNA polymerase (Nature)
- Organelle isolation: functional mitochondria from mouse liver, muscle and cultured filroblasts (Nature Protocols)
- Disruption of Fusion Results in Mitochondrial Heterogeneity and Dysfunction* (Journal of Biological Chemistry)
- Mitochondrial DNA deletions are abundant and cause functional impairment in aged human substantia nigra neurons (Nature Genetics)
- High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease (Nature Genetics)
- Mitochondrial respiratory-chain diseases. (New England Journal of Medicine)
- Transcriptional co-activator PGC-1α drives the formation of slow-twitch muscle fibres (Nature)
- The machines that divide and fuse mitochondria (Annual Review of Biochemistry)
- Mitochondrial threshold effects. (Biochemical Journal)
- MITOCHONDRIAL DNA MUTATIONS IN HUMAN DISEASE (Nature reviews genetics)
Their papers
- Mitochondrial DNA Mutations, Oxidative Stress, and Apoptosis in Mammalian Aging (2005) — 2,111 citations
- Mitochondrial fusion is required for mtDNA stability in skeletal muscle and tolerance of mtDNA mutations (2010) — 1,194 citations
- A mixture model approach for the analysis of microarray gene expression data (2002) — 390 citations
- Sirt3 Mediates Reduction of Oxidative Damage and Prevention of Age-related Hearing Loss under Caloric Restriction (2010) — 1,156 citations
- DNA damage-induced alterations in chromatin contribute to genomic integrity and age-related changes in gene expression (2008) — 791 citations
- Calorie restriction and SIRT3 trigger global reprogramming of the mitochondrial protein acetylome. (2013) — 643 citations
- A Low Dose of Dietary Resveratrol Partially Mimics Caloric Restriction and Retards Aging Parameters in Mice (2008) — 618 citations
- SIRT4 inhibits glutamate dehydrogenase and opposes the effects of calorie restriction in pancreatic beta cells. (2006) — 1,197 citations
- The selenoprotein GPX4 is essential for mouse development and protects from radiation and oxidative damage insults. (2003) — 760 citations
- Gene-expression profile of the ageing brain in mice (2000) — 1,085 citations