Bennett G. Childs

Physiology · 2011–2017

A researcher in the Science corpus: 6 papers; trusts 4 researchers and is trusted by 6 researchers here, as inferred from the citation graph.

Subfields

Whom Bennett G. Childs trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. D. Baker
  2. Jean-Philippe Coppé
  3. T. Kuilman
  4. J. Campisi
  5. V. Krizhanovsky
  6. S. Parrinello
  7. R. Kortlever
  8. Jan M. van Deursen
  9. D. Peeper
  10. A. Krtolica

Who trusts Bennett G. Childs overall

  1. Alejandra Hernandez-Segura
  2. I. Tabas
  3. M. Demaria
  4. S. Glück
  5. Jesús Gil
  6. J. Kirkland
  7. R. Kumari
  8. Jodie Birch
  9. Akiko Takahashi
  10. Shenghui He

Papers from Bennett G. Childs's perspective

Ordered by how strongly the researchers Bennett G. Childs trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis (Nature)
  2. Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
  3. Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence (Nature Cell Biology)
  4. Chemokine signaling via the CXCR2 receptor reinforces senescence. (Cell)
  5. Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (PLoS Biology)
  6. Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
  7. Oncogene-induced senescence as an initial barrier in lymphoma development (Nature)
  8. Senescence surveillance of pre-malignant hepatocytes limits liver cancer development (Nature)
  9. Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
  10. Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)

Their papers