S. Glück
Molecular Biology · 2017–2023
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 37 citations into it
- Physiology — 31 citations into it
Whom S. Glück trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Adam Freund
- Jan M. van Deursen
- G. Barber
- Bennett G. Childs
- Qi Chen
- J. Campisi
- C. López‐Otín
- Lijun Sun
- V. Krizhanovsky
- Daxing Gao
Papers from S. Glück's perspective
Ordered by how strongly the researchers S. Glück trusts endorse each paper. Same corpus, a different order for every researcher.
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Chemokine signaling via the CXCR2 receptor reinforces senescence. (Cell)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Lamin B1 loss is a senescence-associated biomarker (Molecular Biology of the Cell)
- p38MAPK is a novel DNA damage response‐independent regulator of the senescence‐associated secretory phenotype (EMBO Journal)
- Cellular senescence in aging and age-related disease: from mechanisms to therapy (Nature Medicine)
- Cyclic GMP-AMP Containing Mixed Phosphodiester Linkages Is An Endogenous High Affinity Ligand for STING (Molecules and Cells)
- Activation of cyclic GMP-AMP synthase by self-DNA causes autoimmune diseases (Proceedings of the National Academy of Sciences of the United States of America)
- Regulation and function of the cGAS–STING pathway of cytosolic DNA sensing
- cGAS produces a 2′-5′-linked cyclic dinucleotide second messenger that activates STING (Nature)
Their papers
- cGAS–STING drives ageing-related inflammation and neurodegeneration (2023) — 793 citations
- Innate immune sensing of cytosolic chromatin fragments through cGAS promotes senescence (2017) — 1,037 citations