Senescence and apoptosis: dueling or complementary cell fates?
By Bennett G. Childs, D. Baker, J. Kirkland, J. Campisi, J. Deursen. EMBO Reports, 2014
803 citations recorded by the source corpus.
Subfields: Aging, Molecular Biology, Physiology
View Paper: https://www.semanticscholar.org/paper/6dcae32219a58a7c48ea560350d65fa0e3b88961
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Aging
In Molecular Biology
- Alejandra Hernandez-Segura
- J. Nehme
- Akiko Takahashi
- Sugiko Watanabe
- M. Takasugi
- Masato T. Kanemaki
- C. Obuse
- Yuka Kawamata
In Physiology
- Alejandra Hernandez-Segura
- J. Nehme
- Akiko Takahashi
- Sugiko Watanabe
- M. Takasugi
- Masato T. Kanemaki
- C. Obuse
- Yuka Kawamata
Citations in the corpus
- Hallmarks of Cellular Senescence. (2018, positive): “Apoptosis Resistance Senescent cells activate several prosurvival factors and become resistant to apoptosis [81].”
- Exosomes maintain cellular homeostasis by excreting harmful DNA from cells (2017, positive): “Since the accumulation of DNA damage is known to cause apoptosis or cellular senescence, depending on the degree of DNA damage 1–3, 41 , we tested whether the Exosome secretion prevents aberrant innate immune response .”