M. Löbrich
Molecular Biology · 2003–2010
A researcher in the Science corpus: 7 papers; trusts 8 researchers and is trusted by 13 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 72 citations into it; #4,374 of 17,773 by trust here, #4,213 by citations
- Oncology — 39 citations into it; #960 of 1,698 by trust here, #638 by citations
- Cancer Research — 22 citations into it; #422 of 1,780 by trust here, #605 by citations
- Cell Biology — 16 citations into it
Whom M. Löbrich trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- J. Bartek
- K. Rothkamm
- J. Lukas
- Jacob C. Harrison
- J. Melo
- Stephen J. Elledge
- Yosef Shiloh
- F. Mitelman
- Jan H. J. Hoeijmakers
Who trusts M. Löbrich overall
Papers from M. Löbrich's perspective
Ordered by how strongly the researchers M. Löbrich trusts endorse each paper. Same corpus, a different order for every researcher.
- Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks (Nature Cell Biology)
- MDC1 is a mediator of the mammalian DNA damage checkpoint (Nature)
- DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis (Nature)
- Chromatin relaxation in response to DNA double-strand breaks is modulated by a novel ATM- and KAP-1 dependent pathway (Nature Cell Biology)
- A silencing pathway to induce H3-K9 and H4-K20 trimethylation at constitutive heterochromatin. (Genes & Development)
- Mammalian G1- and S-phase checkpoints in response to DNA damage. (Current Opinion in Cell Biology)
- The impact of translocations and gene fusions on cancer causation (Nature Reviews. Cancer)
- Cooperation between complexes that regulate chromatin structure and transcription. (Cell)
- Suv39h-mediated histone H3 lysine 9 methylation directs DNA methylation to major satellite repeats at pericentric heterochromatin. (Current Biology)
- Surviving the breakup: the DNA damage checkpoint. (Annual Review of Genetics)
Their papers
- Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses (2003) — 1,724 citations
- Pathways of DNA Double-Strand Break Repair during the Mammalian Cell Cycle (2003) — 1,253 citations
- ATM and DNA-PK Function Redundantly to Phosphorylate H2AX after Exposure to Ionizing Radiation (2004) — 1,057 citations
- A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci. (2004) — 921 citations
- ATM signaling facilitates repair of DNA double-strand breaks associated with heterochromatin. (2008) — 902 citations
- The impact of a negligent G2/M checkpoint on genomic instability and cancer induction (2007) — 640 citations
- γH2AX foci analysis for monitoring DNA double-strand break repair: Strengths, limitations and optimization (2010) — 637 citations