S. Magesh
Molecular Biology · 2012
A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 40 citations into it
Whom S. Magesh trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Donna D. Zhang
- A. Dinkova-Kostova
- T. Ozben
- N. Wakabayashi
- A. Kobayashi
- Masayuki Yamamoto
- Makoto Kobayashi
- Gerasimos P. Sykiotis
- Liam Baird
- Wenge Li
Who trusts S. Magesh overall
Papers from S. Magesh's perspective
Ordered by how strongly the researchers S. Magesh trusts endorse each paper. Same corpus, a different order for every researcher.
- Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
- Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway. (Annual Review of Pharmacology and Toxicology)
- Direct evidence that sulfhydryl groups of Keap1 are the sensors regulating induction of phase 2 enzymes that protect against carcinogens and oxidants (Proceedings of the National Academy of Sciences of the United States of America)
- Protection against electrophile and oxidant stress by induction of the phase 2 response: Fate of cysteines of the Keap1 sensor modified by inducers (Proceedings of the National Academy of Sciences of the United States of America)
- Oxidative and Electrophilic Stresses Activate Nrf2 through Inhibition of Ubiquitination Activity of Keap1 (Molecular and Cellular Biology)
- Molecular Mechanisms Activating the Nrf2-Keap1 Pathway of Antioxidant Gene Regulation (Antioxidants and Redox Signaling)
- Mechanistic Studies of the Nrf2-Keap1 Signaling Pathway (Drug metabolism reviews (Softcover ed.))
- Molecular mechanisms of the Keap1–Nrf2 pathway in stress response and cancer evolution (Genes to Cells)
- Chronic inflammation and oxidative stress in human carcinogenesis (International Journal of Cancer)
- An important role of Nrf2-ARE pathway in the cellular defense mechanism. (Journal of biochemistry and molecular biology)