Gerasimos P. Sykiotis

Molecular Biology · 2008–2019

A researcher in the Science corpus: 3 papers; trusts 14 researchers and is trusted by 5 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 69 citations into it; #7,697 of 17,773 by trust here, #11,608 by citations
• Immunology — 16 citations into it
• Aging — 11 citations into it; #91 of 261 by trust here, #231 by citations
• Physiology — 11 citations into it
• Endocrine and Autonomic Systems — 9 citations into it
• Health, Toxicology and Mutagenesis — 8 citations into it
• Plant Science — 8 citations into it
• Organic Chemistry — 5 citations into it

Whom Gerasimos P. Sykiotis trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Joy S Wu
2. Donna D. Zhang
3. Sean E. McGuire
4. Hozumi Motohashi
5. Masayuki Yamamoto
6. K. Itoh
7. Norbert Perrimon
8. Liqun Luo
9. K. I. Tong
10. J. An

Who trusts Gerasimos P. Sykiotis overall

1. James W. Kaspar
2. Suresh Niture
3. Q. Ma
4. Anil K. Jaiswal
5. Liam Baird
6. Vy Ngo
7. S. Magesh
8. Pranavi Koppula

Papers from Gerasimos P. Sykiotis's perspective

Ordered by how strongly the researchers Gerasimos P. Sykiotis trusts endorse each paper. Same corpus, a different order for every researcher.

1. Nrf2-Keap1 defines a physiologically important stress response mechanism. (Trends in Molecular Medicine)
2. Molecular mechanism activating Nrf2-Keap1 pathway in regulation of adaptive response to electrophiles. (Free Radical Biology & Medicine)
3. Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer. (Molecules and Cells)
4. Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation (Nature Genetics)
5. Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy (Proceedings of the National Academy of Sciences of the United States of America)
6. Nrf2 Is a Direct PERK Substrate and Effector of PERK-Dependent Cell Survival (Molecular and Cellular Biology)
7. Targeted gene expression as a means of altering cell fates and generating dominant phenotypes.
8. Two neurons mediate diet-restriction-induced longevity in C. elegans (Nature)
9. PERK-dependent Activation of Nrf2 Contributes to Redox Homeostasis and Cell Survival following Endoplasmic Reticulum Stress* (Journal of Biological Chemistry)
10. SKN-1 links C. elegans mesendodermal specification to a conserved oxidative stress response. (Genes & Development)

Their papers

• Stress-Activated Cap’n’collar Transcription Factors in Aging and Human Disease (2010) — 775 citations
• Keap1/Nrf2 signaling regulates oxidative stress tolerance and lifespan in Drosophila. (2008) — 662 citations
• Adverse effects of immune-checkpoint inhibitors: epidemiology, management and surveillance (2019) — 1,924 citations