Melba C. Jaramillo

Molecular Biology · 2013

A researcher in the Science corpus: 1 paper; trusts 4 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

Whom Melba C. Jaramillo trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Donna D. Zhang
  2. T. Kensler
  3. N. Wakabayashi
  4. Masayuki Yamamoto
  5. Hozumi Motohashi
  6. M. Furukawa
  7. Xiao-jun Wang
  8. S. Magesh
  9. A. Kobayashi

Who trusts Melba C. Jaramillo overall

  1. Navdeep S. Chandel
  2. Ralph J. Deberardinis
  3. Lucas B. Sullivan
  4. Ankita Bansal
  5. Brent R. Stockwell
  6. Feng He

Papers from Melba C. Jaramillo's perspective

Ordered by how strongly the researchers Melba C. Jaramillo trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway. (Annual Review of Pharmacology and Toxicology)
  2. Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
  3. Direct evidence that sulfhydryl groups of Keap1 are the sensors regulating induction of phase 2 enzymes that protect against carcinogens and oxidants (Proceedings of the National Academy of Sciences of the United States of America)
  4. BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase (Molecular and Cellular Biology)
  5. Distinct Cysteine Residues in Keap1 Are Required for Keap1-Dependent Ubiquitination of Nrf2 and for Stabilization of Nrf2 by Chemopreventive Agents and Oxidative Stress (Molecular and Cellular Biology)
  6. Loss of Keap1 function activates Nrf2 and provides advantages for lung cancer cell growth. (Cancer Research)
  7. Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer. (Molecules and Cells)
  8. Direct interaction between Nrf2 and p21(Cip1/WAF1) upregulates the Nrf2-mediated antioxidant response. (Molecules and Cells)
  9. The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (Nature Cell Biology)
  10. Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis (Nature)

Their papers