P. Pham
Molecular Biology · 2003
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 10 citations into it; #13,904 of 17,773 by trust here, #10,695 by citations
Whom P. Pham trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts P. Pham overall
Papers from P. Pham's perspective
Ordered by how strongly the researchers P. Pham trusts endorse each paper. Same corpus, a different order for every researcher.
- Transcription-targeted DNA deamination by the AID antibody diversification enzyme (Nature)
- Activation-induced cytidine deaminase deaminates deoxycytidine on single-stranded DNA but requires the action of RNase (Proceedings of the National Academy of Sciences of the United States of America)
- AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversification (Nature)
- Error-prone repair DNA polymerases in prokaryotes and eukaryotes. (Annual Review of Biochemistry)
- Immunoglobulin isotype switching is inhibited and somatic hypermutation perturbed in UNG-deficient mice. (Current Biology)
- Molecular mechanism of class switch recombination: linkage with somatic hypermutation. (Annual Review of Immunology)
- Faculty Opinions recommendation of Isolation of a human gene that inhibits HIV-1 infection and is suppressed by the viral Vif protein.
- Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme. (Cell)
- Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2). (Cell)
- The mechanism and regulation of chromosomal V(D)J recombination. (Cell)