S. Shoelson

Epidemiology · 2001–2012

A researcher in the Science corpus: 13 papers; trusts 10 researchers and is trusted by 25 researchers here, as inferred from the citation graph.

Subfields

• Epidemiology — 103 citations into it; #111 of 2,148 by trust here, #36 by citations
• Physiology — 72 citations into it; #524 of 2,276 by trust here, #151 by citations
• Immunology — 49 citations into it; #591 of 2,160 by trust here, #475 by citations
• Molecular Biology — 39 citations into it; #9,159 of 17,773 by trust here, #3,737 by citations
• Cancer Research — 12 citations into it; #1,101 of 1,780 by trust here, #245 by citations

Whom S. Shoelson trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. S. Weisberg
2. M. Yuan
3. G. Hotamışlıgil
4. S. Summers
5. C. Lumeng
6. D. Cai

Who trusts S. Shoelson overall

1. Jerrold M. Olefsky
2. Christopher K. Glass
3. Jun Yoshino
4. Haiming Cao
5. G. Targher
6. Jianping Ye
7. J. Odegaard
8. Herbert Tilg
9. Brandon H Hidaka
10. Christopher D. Byrne

Papers from S. Shoelson's perspective

Ordered by how strongly the researchers S. Shoelson trusts endorse each paper. Same corpus, a different order for every researcher.

1. Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. (Journal of Clinical Investigation)
2. Prevention of fat-induced insulin resistance by salicylate. (Journal of Clinical Investigation)
3. C-reactive protein, interleukin 6, and risk of developing type 2 diabetes mellitus. (Journal of the American Medical Association (JAMA))
4. Adipocyte death defines macrophage localization and function in adipose tissue of obese mice and humans Published, JLR Papers in Press, September 8, 2005. DOI 10.1194/jlr.M500294-JLR200 (Journal of Lipid Research)
5. Inflammation and insulin resistance. (Diabetes Care)
6. Inflammatory pathways and insulin action (International Journal of Obesity)
7. Fat accumulation in the liver is associated with defects in insulin suppression of glucose production and serum free fatty acids independent of obesity in normal men. (Journal of Clinical Endocrinology and Metabolism)
8. Elevated levels of acute-phase proteins and plasminogen activator inhibitor-1 predict the development of type 2 diabetes: the insulin resistance atherosclerosis study. (Diabetes)
9. A pilot study of pioglitazone treatment for nonalcoholic steatohepatitis (Hepatology)
10. Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair. (Journal of Clinical Investigation)

Their papers

• Prevention of fat-induced insulin resistance by salicylate. (2001) — 745 citations
• Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* (2002) — 1,017 citations
• IKKbeta/NF-kappaB activation causes severe muscle wasting in mice. (2004) — 819 citations
• Mechanism by which high-dose aspirin improves glucose metabolism in type 2 diabetes. (2002) — 708 citations
• PPARγ is a major driver of the accumulation and phenotype of adipose-tissue Treg cells (2012) — 1,109 citations
• Type 2 diabetes as an inflammatory disease (2011) — 3,597 citations
• Fat Treg cells: a liaison between the immune and metabolic systems (2009) — 2,038 citations
• Obesity, inflammation, and insulin resistance. (2007) — 1,843 citations
• Inflammation and insulin resistance. (2006) — 2,561 citations
• Local and systemic insulin resistance resulting from hepatic activation of IKK-beta and NF-kappaB. (2005) — 1,030 citations
• Local and systemic insulin resistance resulting from hepatic activation of IKK-β and NF-κB (2005) — 2,116 citations
• SOCS-1 and SOCS-3 Block Insulin Signaling by Ubiquitin-mediated Degradation of IRS1 and IRS2* (2002) — 941 citations
• Reversal of Obesity- and Diet-Induced Insulin Resistance with Salicylates or Targeted Disruption of Ikkβ (2001) — 2,012 citations