Reversal of Obesity- and Diet-Induced Insulin Resistance with Salicylates or Targeted Disruption of Ikkβ

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Hypoxia-Inducible Factor 1α Induces Fibrosis and Insulin Resistance in White Adipose Tissue (2009, background): “Several components of the inflammatory pathways have been implicated in reducing insulin sensitivity, such as TNF- (21), c-jun N-terminal kinase (19), and NF- B (54).”
Mechanisms of insulin resistance in obesity (2013, background): “Activation of IKKβ and JNK1 involves in IRS-1 inhibition by TNF-α [8,12,13].”
High-Fat Diet: Bacteria Interactions Promote Intestinal Inflammation Which Precedes and Correlates with Obesity and Insulin Resistance in Mouse (2010, background): “TNF-a activates NF-kB and other inflammatory pathways involved in the etiology of insulin resistance and Type 2 diabetes [28,29].”