Inflammation and insulin resistance.

By S. Shoelson, Jongsoon Lee, A. Goldfine. Journal of Clinical Investigation, 2006

2,561 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/e9db8bd9356863d77babce9c10f5bf0d8b552927

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrinology, Diabetes and Metabolism

1. M. Pinzani
2. E. Tsochatzis
3. Jianping Ye
4. E. Buzzetti
5. A. Chait
6. Laura J. den Hartigh

In Epidemiology

1. Jun Yoshino
2. Jamal Naderi
3. M. Longo
4. M. Pinzani
5. E. Tsochatzis
6. Jianping Ye
7. C. Miele
8. G. A. Raciti

In Physiology

1. S. Imai
2. Jun Yoshino
3. Jamal Naderi
4. M. Longo
5. Jianping Ye
6. C. Miele
7. G. A. Raciti
8. F. Beguinot

Citations in the corpus

• Chronic Adipose Tissue Inflammation Linking Obesity to Insulin Resistance and Type 2 Diabetes (2020, background): “…in the increased expression of several NF-κB target genes [e.g., IL-6, TNFα, interferon-γ (IFNγ), transforming growth factor-β (TGF-β), monocyte chemotactic protein-1 (MCP-1), and interleukin-1β (IL-1β)], which further exacerbate IR progression (Shoelson et al., 2006; Panahi et al., 2018).”
• Adipose Tissue Dysfunction as Determinant of Obesity-Associated Metabolic Complications (2019, background): “, IL-6, TNF-α, interferon gamma (IFN-γ), transforming growth factor beta (TGFβ), MCP-1, and receptor for advanced glycosylation end product (RAGE)] [117,121].”
• Adipose Tissue Distribution, Inflammation and Its Metabolic Consequences, Including Diabetes and Cardiovascular Disease (2020, background): “In contrast with adipose tissue macrophages, hepatic Kupffer cell numbers do not increase with adiposity, but instead become “activated,” akin to M1 or MMe macrophages (308).”