Thomas Schwarz

Molecular Biology · 2002–2012

A researcher in the Science corpus: 5 papers; trusts 8 researchers and is trusted by 14 researchers here, as inferred from the citation graph.

Subfields

Whom Thomas Schwarz trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Koji Okamoto
  2. Derek P. Narendra
  3. T. Kanki
  4. Jian-Sheng Kang
  5. R. Youle

Who trusts Thomas Schwarz overall

  1. N. Hirokawa
  2. Julia J. Harris
  3. Prashant Mishra
  4. D. C. Chan
  5. Shinsuke Niwa

Papers from Thomas Schwarz's perspective

Ordered by how strongly the researchers Thomas Schwarz trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  2. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  3. Essential role for Nix in autophagic maturation of erythroid cells (Nature)
  4. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
  5. Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)
  6. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)
  7. Nix is a selective autophagy receptor for mitochondrial clearance (EMBO Reports)
  8. A Mitochondrial Paradigm of Metabolic and Degenerative Diseases, Aging, and Cancer: A Dawn for Evolutionary Medicine (Annual Review of Genetics)
  9. NIX is required for programmed mitochondrial clearance during reticulocyte maturation (Proceedings of the National Academy of Sciences of the United States of America)
  10. Mitochondria-anchored receptor Atg32 mediates degradation of mitochondria via selective autophagy. (Developmental Cell)

Their papers