Mashun Onishi
Epidemiology · 2021
A researcher in the Science corpus: 1 paper; trusts 5 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Epidemiology — 59 citations into it
- Molecular Biology — 47 citations into it
- Neurology — 25 citations into it
- Cell Biology — 20 citations into it
- Physiology — 18 citations into it
Whom Mashun Onishi trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- D. Selkoe
- J. Lemasters
- Derek P. Narendra
- Y. Wong
- T. Johansen
- Matthew J. Harms
- R. Loomba
- J. Spinelli
- Shireen A. Sarraf
- Koji Okamoto
Papers from Mashun Onishi's perspective
Ordered by how strongly the researchers Mashun Onishi trusts endorse each paper. Same corpus, a different order for every researcher.
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- The mitochondrial deubiquitinase USP30 opposes parkin-mediated mitophagy (Nature)
- Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
- Essential role for Nix in autophagic maturation of erythroid cells (Nature)
- Broad activation of the ubiquitin–proteasome system by Parkin is critical for mitophagy (Human Molecular Genetics)
- Nix is a selective autophagy receptor for mitochondrial clearance (EMBO Reports)
- Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- Molecular mechanisms and physiological functions of mitophagy (2021) — 1,187 citations