M. Vulić
Genetics · 2010
A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Genetics — 12 citations into it; #4,098 of 6,854 by trust here, #4,505 by citations
- Molecular Biology — 10 citations into it; #11,512 of 17,773 by trust here, #13,284 by citations
- Molecular Medicine — 8 citations into it; #167 of 296 by trust here, #243 by citations
Whom M. Vulić trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts M. Vulić overall
Papers from M. Vulić's perspective
Ordered by how strongly the researchers M. Vulić trusts endorse each paper. Same corpus, a different order for every researcher.
- One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products. (Proceedings of the National Academy of Sciences of the United States of America)
- Conditional-Replication, Integration, Excision, and Retrieval Plasmid-Host Systems for Gene Structure-Function Studies of Bacteria (Journal of Bacteriology)
- Biofilms and Planktonic Cells of Pseudomonas aeruginosa Have Similar Resistance to Killing by Antimicrobials (Journal of Bacteriology)
- Bacterial Persistence as a Phenotypic Switch (Science)
- Persister cells and tolerance to antimicrobials. (FEMS Microbiology Letters)
- Construction of Escherichia coli K-12 in-frame, single-gene knockout mutants: the Keio collection (Molecular Systems Biology)
- Specialized Persister Cells and the Mechanism of Multidrug Tolerance in Escherichia coli (Journal of Bacteriology)
- Prokaryotic toxin–antitoxin stress response loci (Nature Reviews Microbiology)
- Antimicrobial peptides of multicellular organisms (Nature)
- A common mechanism of cellular death induced by bactericidal antibiotics. (Cell)
Their papers
- Ciprofloxacin Causes Persister Formation by Inducing the TisB toxin in Escherichia coli (2010) — 770 citations