A. Israël
Cancer Research · 2000–2010
A researcher in the Science corpus: 9 papers; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Cancer Research — 34 citations into it; #789 of 1,780 by trust here, #554 by citations
- Immunology — 28 citations into it; #996 of 2,160 by trust here, #745 by citations
Whom A. Israël trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts A. Israël overall
Papers from A. Israël's perspective
Ordered by how strongly the researchers A. Israël trusts endorse each paper. Same corpus, a different order for every researcher.
- Essential function for the kinase TAK1 in innate and adaptive immune responses (Nature Immunology)
- TAK1, but not TAB1 or TAB2, plays an essential role in multiple signaling pathways in vivo. (Genes & Development)
- TAK1 is a ubiquitin-dependent kinase of MKK and IKK (Nature)
- Signal transduction by tumor necrosis factor and its relatives. (Trends in Cell Biology)
- Signaling to NF-kappaB. (Genes & Development)
- Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity. (Annual Review of Immunology)
- Loss of the cylindromatosis tumour suppressor inhibits apoptosis by activating NF-κB (Nature)
- The TRAF6 ubiquitin ligase and TAK1 kinase mediate IKK activation by BCL10 and MALT1 in T lymphocytes. (Molecules and Cells)
- Toll-like receptor signalling (Nature reviews. Immunology)
- The Lymphotoxin-β Receptor Induces Different Patterns of Gene Expression via Two NF-κB Pathways
Their papers
- X-linked anhidrotic ectodermal dysplasia with immunodeficiency is caused by impaired NF-κB signaling (2001) — 737 citations
- A novel proteolytic cleavage involved in Notch signaling: the role of the disintegrin-metalloprotease TACE. (2000) — 1,203 citations
- The tumour suppressor CYLD negatively regulates NF-κB signalling by deubiquitination (2003) — 1,103 citations
- The IKK complex: an integrator of all signals that activate NF-κB? (2000) — 397 citations
- The Notch ligand Delta1 is sequentially cleaved by an ADAM protease and γ-secretase (2003) — 231 citations
- SCFβ‐TrCP ubiquitin ligase‐mediated processing of NF‐κB p 105 requires phosphorylation of its C‐terminus by IκB kinase (2000) — 167 citations
- The IKK complex, a central regulator of NF-kappaB activation. (2010) — 796 citations
- T-cell-receptor- and B-cell-receptor-mediated activation of NF-κB in lymphocytes (2004) — 120 citations
- Genomic rearrangement in NEMO impairs NF-κB activation and is a cause of incontinentia pigmenti (2000) — 647 citations