L. Sparks

Molecular Biology · 2005–2017

A researcher in the Science corpus: 4 papers; trusts 3 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom L. Sparks trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. John Quackenbush
  2. M. Kerr
  3. Stephen A. Bustin
  4. Trevor J. Hastie
  5. Gary A. Churchill
  6. K. Petersen
  7. Vamsi K. Mootha

Who trusts L. Sparks overall

  1. M. Laplante
  2. W. Sivitz
  3. Nader G. Abraham
  4. David M. Sabatini
  5. M. Yorek
  6. A. Kappas

Papers from L. Sparks's perspective

Ordered by how strongly the researchers L. Sparks trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Coordinated reduction of genes of oxidative metabolism in humans with insulin resistance and diabetes: Potential role of PGC1 and NRF1 (Proceedings of the National Academy of Sciences of the United States of America)
  2. Impaired mitochondrial activity in the insulin-resistant offspring of patients with type 2 diabetes. (New England Journal of Medicine)
  3. Mitochondrial Dysfunction in the Elderly: Possible Role in Insulin Resistance (Science)
  4. Absolute quantification of mRNA using real-time reverse transcription polymerase chain reaction assays. (Journal of Molecular Endocrinology)
  5. Identification of white adipocyte progenitor cells in vivo. (Cell)
  6. Evidence for a role of developmental genes in the origin of obesity and body fat distribution. (Proceedings of the National Academy of Sciences of the United States of America)
  7. Muscle injury activates resident fibro/adipogenic progenitors that facilitate myogenesis (Nature Cell Biology)
  8. Prdm16 determines the thermogenic program of subcutaneous white adipose tissue in mice. (Journal of Clinical Investigation)
  9. 'Gene shaving' as a method for identifying distinct sets of genes with similar expression patterns (Genome Biology)
  10. Dysfunction of mitochondria in human skeletal muscle in type 2 diabetes. (Diabetes)

Their papers