M. Bianchi

Immunology · Nature · 2002–2014

A researcher in the Science corpus: 14 papers; trusts 10 researchers and is trusted by 42 researchers here, as inferred from the citation graph.

Subfields

• Immunology — 97 citations into it; #256 of 2,160 by trust here, #217 by citations
• Molecular Biology — 55 citations into it; #1,167 of 17,773 by trust here, #2,147 by citations
• Clinical Biochemistry — 48 citations into it; #3 of 177 by trust here, #3 by citations
• Epidemiology — 16 citations into it

Whom M. Bianchi trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Paola Scaffidi
2. C. Janeway
3. Fu-Tong Liu
4. S. Seong
5. Tom Misteli
6. M. Ochani
7. U. Andersson
8. Fabio Martinon
9. G. Randolph
10. R. Medzhitov

Who trusts M. Bianchi overall

1. C. Cayrol
2. H. Kono
3. C. Rosales
4. Nikolaos G. Frangogiannis
5. K. Rock
6. J. Klune
7. J. Girard
8. Erika L. Pearce
9. X. Zhang
10. T. Shaw

Papers from M. Bianchi's perspective

Ordered by how strongly the researchers M. Bianchi trusts endorse each paper. Same corpus, a different order for every researcher.

1. Reversing established sepsis with antagonists of endogenous high-mobility group box 1 (Proceedings of the National Academy of Sciences of the United States of America)
2. IMMUNE TOLERANCE INDUCTION BY APOPTOTIC CELLS REQUIRES CASPASE-DEPENDENT OXIDATION OF HMGB1 (Immunity)
3. High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal (Nature reviews. Immunology)
4. Phagocytosis and clearance of apoptotic cells is mediated by MER (Nature)
5. Molecular identification of a danger signal that alerts the immune system to dying cells (Nature)
6. Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion (EMBO Journal)
7. Type i interferons potently enhance humoral immunity and can promote isotype switching by stimulating dendritic cells in vivo. (Immunity)
8. A blast from the past: clearance of apoptotic cells regulates immune responses (Nature reviews. Immunology)
9. Consequences of Cell Death (Journal of Experimental Medicine)
10. Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-kappa B pathway. (International Immunology)

Their papers

• Release of chromatin protein HMGB1 by necrotic cells triggers inflammation (2002) — 4,107 citations
• DAMPs, PAMPs and alarmins: all we need to know about danger (2007) — 2,915 citations
• Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion (2003) — 1,317 citations
• Endogenous HMGB1 regulates autophagy (2010) — 936 citations
• High-Mobility Group Box-1 in Ischemia-Reperfusion Injury of the Heart (2008) — 620 citations
• Mutually exclusive redox forms of HMGB1 promote cell recruitment or proinflammatory cytokine release (2012) — 726 citations
• Ultraviolet-radiation-induced inflammation promotes angiotropism and metastasis in melanoma (2014) — 603 citations
• Cancer cell–autonomous contribution of type I interferon signaling to the efficacy of chemotherapy (2014) — 997 citations
• HMGB1 promotes recruitment of inflammatory cells to damaged tissues by forming a complex with CXCL12 and signaling via CXCR4 (2012) — 644 citations
• Toll-like receptor 4 and high-mobility group box-1 are involved in ictogenesis and can be targeted to reduce seizures (2010) — 897 citations
• HMGB proteins function as universal sentinels for nucleic-acid-mediated innate immune responses (2009) — 719 citations
• High‐mobility group box 1 (HMGB1) protein at the crossroads between innate and adaptive immunity (2007) — 606 citations
• HMGB1 is an endogenous immune adjuvant released by necrotic cells (2004) — 622 citations
• The nuclear protein HMGB1 is secreted by monocytes via a non‐classical, vesicle‐mediated secretory pathway (2002) — 988 citations