High‐mobility group box 1 (HMGB1) protein at the crossroads between innate and adaptive immunity

By M. Bianchi, A. Manfredi. Immunological Reviews, 2007

606 citations recorded by the source corpus.

Subfields: Clinical Biochemistry, Immunology

View Paper: https://www.semanticscholar.org/paper/71d98f0e4cde391dd3d3bffbb3946defd3594cc4

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Clinical Biochemistry

1. R. Dhupar
2. Jon S. Cardinal
3. J. Klune
4. A. Tsung
5. Timothy R. Billiar

In Immunology

1. Guido Kroemer
2. L. Zitvogel
3. John V. Forrester
4. Douglas R. Green
5. T. Ferguson
6. Mei Chen
7. Heping Xu
8. L. Galluzzi

Citations in the corpus

• Immunogenic and tolerogenic cell death (2009, background): “It seems that HMGB1 can bind several pattern recognition receptors (PRRs), including Toll-like receptor 2 ( TLR2 ), TLR4 and RAGE (receptor for advanced glycosylation end products; also known as AGER”
• Para-inflammation in the aging retina. (2009, background): “Receptors for alarmins include the interleukin (IL)-1 receptor, RAGEs (Bianchi and Manfredi, 2007), receptor-type tyrosine phosphatase (RPTP), and TLRs (Miyake, 2007; Zhang and Schluesener, 2006).”
• HMGB1: Endogenous Danger Signaling (2008, background): “REGENERATIVE EFFECTS OF HMGB1 In contrast to their role in promoting inflammation, the ability of alarmins to promote tissue repair and regeneration is of increasing interest (73).”