Anil K. Jaiswal
Molecular Biology · 2004–2013
A researcher in the Science corpus: 4 papers; trusts 7 researchers and is trusted by 6 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 62 citations into it; #4,622 of 17,773 by trust here, #7,911 by citations
- Plant Science — 7 citations into it; #1,948 of 8,232 by trust here, #6,797 by citations
- Biochemistry — 6 citations into it
- Organic Chemistry — 5 citations into it
Whom Anil K. Jaiswal trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Anil K. Jaiswal overall
Papers from Anil K. Jaiswal's perspective
Ordered by how strongly the researchers Anil K. Jaiswal trusts endorse each paper. Same corpus, a different order for every researcher.
- Distinct Cysteine Residues in Keap1 Are Required for Keap1-Dependent Ubiquitination of Nrf2 and for Stabilization of Nrf2 by Chemopreventive Agents and Oxidative Stress (Molecular and Cellular Biology)
- Enhanced Expression of the Transcription Factor Nrf2 by Cancer Chemopreventive Agents: Role of Antioxidant Response Element-Like Sequences in the nrf2 Promoter (Molecular and Cellular Biology)
- Keap1 regulates both cytoplasmic‐nuclear shuttling and degradation of Nrf2 in response to electrophiles (Genes to Cells)
- Loss of Keap1 function activates Nrf2 and provides advantages for lung cancer cell growth. (Cancer Research)
- Direct interaction between Nrf2 and p21(Cip1/WAF1) upregulates the Nrf2-mediated antioxidant response. (Molecules and Cells)
- Keap1-dependent Proteasomal Degradation of Transcription Factor Nrf2 Contributes to the Negative Regulation of Antioxidant Response Element-driven Gene Expression* (Journal of Biological Chemistry)
- The Keap1-BTB Protein Is an Adaptor That Bridges Nrf2 to a Cul3-Based E3 Ligase: Oxidative Stress Sensing by a Cul3-Keap1 Ligase (Molecular and Cellular Biology)
- Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy (Proceedings of the National Academy of Sciences of the United States of America)
- Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis (Nature)
- Nrf2-Keap1 regulation of cellular defense mechanisms against electrophiles and reactive oxygen species. (Advances in Enzyme Regulation)
Their papers
- Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid. (2004) — 763 citations
- Nrf2 signaling in coordinated activation of antioxidant gene expression. (2004) — 1,296 citations
- Regulation of Nrf2 – An update (2013) — 854 citations
- Nrf2:INrf2(Keap1) Signaling in Oxidative Stress (2009) — 1,592 citations