L. Steinman

Immunology · 2000–2022

A researcher in the Science corpus: 11 papers; trusts 1 researcher and is trusted by 7 researchers here, as inferred from the citation graph.

Subfields

Whom L. Steinman trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Yi Chen
  2. E. Bettelli
  3. I. Ivanov

Who trusts L. Steinman overall

  1. C. Dong
  2. C. Iadecola
  3. B. Trapp
  4. Daniel S. Reich
  5. J. Goverman
  6. S. Gaffen
  7. R. Straub

Papers from L. Steinman's perspective

Ordered by how strongly the researchers L. Steinman trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation (Journal of Experimental Medicine)
  2. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
  3. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  4. A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17 (Nature Immunology)
  5. Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
  6. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
  7. A crucial role for interleukin (IL)-1 in the induction of IL-17–producing T cells that mediate autoimmune encephalomyelitis (Journal of Experimental Medicine)
  8. Anti-IL-23 therapy inhibits multiple inflammatory pathways and ameliorates autoimmune encephalomyelitis. (Journal of Clinical Investigation)
  9. Anti-inflammatory activity of IVIG mediated through the inhibitory Fc receptor. (Science)
  10. The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells. (Cell)

Their papers