Andrew N. J. McKenzie

Immunology · 2006–2018

A researcher in the Science corpus: 12 papers; trusts 3 researchers and is trusted by 6 researchers here, as inferred from the citation graph.

Subfields

Whom Andrew N. J. McKenzie trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. H. Spits (in Immunology)
  2. L. Lanier (in Immunology)
  3. D. Artis (in Immunology)

Who trusts Andrew N. J. McKenzie

  1. D. Artis (in Immunology)
  2. J. Girard (in Immunology)
  3. H. Hammad (in Immunology)
  4. B. Lambrecht (in Immunology)
  5. H. Spits (in Immunology)
  6. C. Cayrol (in Immunology)

Papers from Andrew N. J. McKenzie's perspective

Ordered by how strongly the researchers Andrew N. J. McKenzie trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Nuocytes represent a new innate effector leukocyte that mediates type-2 immunity (Nature)
  2. Systemically dispersed innate IL-13–expressing cells in type 2 immunity (Proceedings of the National Academy of Sciences of the United States of America)
  3. Innate lymphoid cells promote lung-tissue homeostasis after infection with influenza virus (Nature Immunology)
  4. Lung natural helper cells are a critical source of Th2 cell-type cytokines in protease allergen-induced airway inflammation. (Immunity)
  5. Lymphoid tissue genesis induced by commensals through NOD1 regulates intestinal homeostasis (Nature)
  6. Interleukin-22 but not interleukin-17 provides protection to hepatocytes during acute liver inflammation. (Immunity)
  7. Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides (Journal of Experimental Medicine)
  8. Innate and adaptive interleukin-22 protects mice from inflammatory bowel disease (Immunity)
  9. IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. (Immunity)
  10. Type 2 innate lymphoid cells control eosinophil homeostasis (Nature)

Their papers