Innate lymphoid cells — how did we miss them?
By Jennifer A Walker, J. Barlow, Andrew N. J. McKenzie. Nature reviews. Immunology, 2013
703 citations recorded by the source corpus.
Subfields: Immunology, Surgery
View Paper: https://www.semanticscholar.org/paper/2d040624b1636970743dec273b9fb4eab27799ac
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Citations in the corpus
- Activated Type 2 Innate Lymphoid Cells regulate Beige Fat Biogenesis (2014, positive): “or IL-13 (Koyasu and Moro, 2013; McKenzie et al., 2014; Walker et al., 2013), directly stimulate the proliferation of PDGFRa APs.”
- Chemokines and chemokine receptors: positioning cells for host defense and immunity. (2014, background): “ILC2 express CXCR4 as well as CXCR6 and CCR9, which promote their homeostatic distribution in the skin and gut, respectively (71, 72).”
- The chemokine system in innate immunity. (2015, background): “ILC2 express CXCR4 and CCR9, which promote their homeostatic distribution (Walker et al. 2013).”
- Cellular and molecular mechanisms of skin wound healing (2024, neutral): “Innate lymphoid cells (ILCs) are a family of innate cells that do not express antigen-specific receptors 200 .”
- Structure-function of the immune system in the spleen (2019, background): “TH2-like cytokines—IL-5, IL-9, and IL-13— are produced by ILC2s found in the spleen (72).”
- TSLP elicits IL-33–independent innate lymphoid cell responses to promote skin inflammation (2013, background): “2B), similar to group 2 ILCs previously identified in the intestine, lymphoid tissue, and lung of mice (15).”