J. Harper
Molecular Biology · Molecules and Cells · 2000–2020
A researcher in the Science corpus: 22 papers; trusts 5 researchers and is trusted by 19 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 288 citations into it; #3,102 of 17,773 by trust here, #1,385 by citations
- Epidemiology — 157 citations into it; #589 of 2,148 by trust here, #303 by citations
- Oncology — 48 citations into it; #217 of 1,698 by trust here, #355 by citations
- Neurology — 36 citations into it; #1,110 of 1,455 by trust here, #517 by citations
Whom J. Harper trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts J. Harper overall
- D. Hanahan
- Kevin M. Prise
- Ji Luo
- R. Weinberg
- J. O’Sullivan
- Stephen J. Elledge
- N. Solimini
- D. Komander
- Stephen P. Jackson
- Alberto Ciccia
Papers from J. Harper's perspective
Ordered by how strongly the researchers J. Harper trusts endorse each paper. Same corpus, a different order for every researcher.
- RNF8 ubiquitylates histones at DNA double-strand breaks and promotes assembly of repair proteins. (Cell)
- Mechanisms of mitophagy (Nature reviews. Molecular cell biology)
- RNF8 transduces the DNA-damage signal via histone ubiquitylation and checkpoint protein assembly. (Cell)
- Sensing DNA Damage Through ATRIP Recognition of RPA-ssDNA Complexes (Science)
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- TopBP1 activates the ATR-ATRIP complex. (Cell)
- DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation (Nature)
- Quantitative Phosphoproteomics Reveals Widespread Full Phosphorylation Site Occupancy During Mitosis (Science Signaling)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- The mitochondrial deubiquitinase USP30 opposes parkin-mediated mitophagy (Nature)
Their papers
- A Map of the Interactome Network of the Metazoan C. elegans (2004) — 1,796 citations
- Defining the human deubiquitinating enzyme interaction landscape. (2009) — 1,568 citations
- Structure of a -TrCP1-Skp1--Catenin Complex (2003) — 523 citations
- The DNA damage response: ten years after. (2007) — 1,755 citations
- The BioPlex Network: A Systematic Exploration of the Human Interactome (2015) — 1,351 citations
- Quantitative proteomics reveal a feed-forward model for mitochondrial PARKIN translocation and UB chain synthesis (2014) — 664 citations
- Phosphorylation-Dependent Ubiquitination of Cyclin E by the SCFFbw7 Ubiquitin Ligase (2001) — 845 citations
- Systematic analysis and nomenclature of mammalian F-box proteins. (2004) — 722 citations
- Insights into SCF ubiquitin ligases from the structure of the Skp1–Skp2 complex (2000) — 637 citations
- Systematic and quantitative assessment of the ubiquitin modified proteome (2011) — 1,651 citations
- Drug discovery in the ubiquitin–proteasome system (2006) — 604 citations
- Ubiquitin-like protein activation by E1 enzymes: the apex for downstream signalling pathways (2009) — 940 citations
- Dual Proteome-scale Networks Reveal Cell-specific Remodeling of the Human Interactome (2020) — 824 citations
- Molecular definitions of autophagy and related processes (2017) — 1,459 citations
- Architecture of the human interactome defines protein communities and disease networks (2017) — 1,317 citations
- Editorial :Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition). DOI: 10.1080/15548627.2015.1100356;WOS:000373595400001; 2-s2.0-85013763791&;PMID: 26799652 (2016) — 1,523 citations
- Exome sequencing in amyotrophic lateral sclerosis identifies risk genes and pathways (2015) — 948 citations
- The PINK1-PARKIN mitochondrial ubiquitylation pathway drives a program of TBK1 activation and recruitment of OPTN and NDP52 to promote mitophagy (2015) — 803 citations
- Quantitative proteomics identifies NCOA4 as the cargo receptor mediating ferritinophagy (2014) — 1,845 citations
- A family of diverse Cul4-Ddb1-interacting proteins includes Cdt2, which is required for S phase destruction of the replication factor Cdt1. (2006) — 647 citations