E. Masliah
Physiology · 2000–2019
A researcher in the Science corpus: 30 papers; trusts 6 researchers and is trusted by 6 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 260 citations into it; #867 of 2,276 by trust here, #34 by citations
- Neurology — 220 citations into it; #813 of 1,455 by trust here, #14 by citations
- Molecular Biology — 166 citations into it; #12,698 of 17,773 by trust here, #2,317 by citations
- Cellular and Molecular Neuroscience — 112 citations into it; #2,657 of 2,998 by trust here, #132 by citations
- Psychiatry and Mental health — 106 citations into it; #1,203 of 1,338 by trust here, #96 by citations
- Genetics — 82 citations into it
- Cognitive Neuroscience — 35 citations into it
- Pharmacology — 29 citations into it
Whom E. Masliah trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts E. Masliah overall
Papers from E. Masliah's perspective
Ordered by how strongly the researchers E. Masliah trusts endorse each paper. Same corpus, a different order for every researcher.
- Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis (Science)
- Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
- Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's disease. (Neuron)
- Pathways towards and away from Alzheimer's disease (Nature)
- Stages of the Pathologic Process in Alzheimer Disease: Age Categories From 1 to 100 Years (Journal of Neuropathology and Experimental Neurology)
- LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing (EMBO Journal)
- Neuropathology of Cognitively Normal Elderly (Journal of Neuropathology and Experimental Neurology)
- Aggresomes, inclusion bodies and protein aggregation. (Trends in Cell Biology)
- Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function (Science)
- Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior. (Behavioural Brain Research)
Their papers
- High-Level Neuronal Expression of Aβ1–42 in Wild-Type Human Amyloid Protein Precursor Transgenic Mice: Synaptotoxicity without Plaque Formation (2000) — 1,924 citations
- S-Nitrosylated protein-disulphide isomerase links protein misfolding to neurodegeneration (2006) — 899 citations
- Altered expression of synaptic proteins occurs early during progression of Alzheimer’s disease (2001) — 713 citations
- A Compendium of Promoter-Centered Long-Range Chromatin Interactions in the Human Genome (2019) — 319 citations
- Common variants in MS4A4/MS4A6E, CD2uAP, CD33, and EPHA1 are associated with late-onset Alzheimer’s disease (2011) — 1,887 citations
- Axonopathy and Transport Deficits Early in the Pathogenesis of Alzheimer's Disease (2005) — 1,227 citations
- Genetic meta-analysis of diagnosed Alzheimer’s disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing (2019) — 2,422 citations
- Acetylation of tau inhibits its degradation and contributes to tauopathy. (2010) — 725 citations
- Diagnosis and management of dementia with Lewy bodies (2017) — 2,290 citations
- Primary age-related tauopathy (PART): a common pathology associated with human aging (2014) — 1,348 citations
- Neuron-released oligomeric α-synuclein is an endogenous agonist of TLR2 for paracrine activation of microglia (2013) — 813 citations
- Inhibitory Interneuron Deficit Links Altered Network Activity and Cognitive Dysfunction in Alzheimer Model (2012) — 1,160 citations
- National Institute on Aging–Alzheimer’s Association guidelines for the neuropathologic assessment of Alzheimer’s disease (2012) — 2,830 citations
- The many faces of α-synuclein: from structure and toxicity to therapeutic target (2012) — 1,544 citations
- Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature (2012) — 1,882 citations
- National Institute on Aging-Alzheimer’s Association guidelines for the neuropathologic assessment of Alzheimer’s disease: a practical approach (2011) — 2,639 citations
- Neuropathological alterations in Alzheimer disease. (2011) — 3,148 citations
- In vivo demonstration that α-synuclein oligomers are toxic (2011) — 1,193 citations
- Molecular mechanisms of neurodegeneration in Alzheimer's disease. (2010) — 721 citations
- Direct Transfer of α-Synuclein from Neuron to Astroglia Causes Inflammatory Responses in Synucleinopathies* (2010) — 775 citations