Molecular mechanisms of neurodegeneration in Alzheimer's disease.

By Leslie A. Crews, E. Masliah. Human Molecular Genetics, 2010

721 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Developmental Neuroscience, Physiology

View Paper: https://www.semanticscholar.org/paper/55f85a9e4b95b2b7808ce64be21e079f5e16bcbd

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. E. Masliah
2. Bradley T. Hyman
3. Matthew P. Frosch

In Developmental Neuroscience

1. E. Masliah

In Physiology

1. A. Serrano‐Pozo
2. E. Masliah
3. Bradley T. Hyman
4. Matthew P. Frosch

Citations in the corpus

• Neuropathological alterations in Alzheimer disease. (2011, background): “Classical positive lesions consist of abundant amyloid plaques and neurofibrillary tangles, neuropil threads, and dystrophic neurites containing hyperphosphorylated tau (see Box 1 for glossary) (Terry et al. 1994, Mandelkow and Mandelkow 1998, Trojanowski and Lee 2000; Iqbal and Grundke-Iqbal 2002; ”
• Alzheimer’s disease: pathogenesis, diagnostics, and therapeutics (2019, background): “Aβ then oligomerizes, diffuses into synaptic clefts, and interferes with synaptic signaling.(11,12) Consequently, it polymerizes into insoluble amyloid fibrils that aggregate into plaques.”