D. Muñoz‐Espín
Physiology · 2013–2014
A researcher in the Science corpus: 2 papers; trusts 5 researchers and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Physiology — 58 citations into it; #1,772 of 2,276 by trust here, #776 by citations
- Molecular Biology — 50 citations into it; #16,173 of 17,773 by trust here, #10,193 by citations
- Immunology — 20 citations into it
Whom D. Muñoz‐Espín trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- M. Collado
- T. Kuilman
- J. Campisi
- Manuel Serrano
- T. Halazonetis
- Karen H. Vousden
- E. L. Greer
- D. Peeper
- R. Flavell
- J. Seoane
Who trusts D. Muñoz‐Espín overall
Papers from D. Muñoz‐Espín's perspective
Ordered by how strongly the researchers D. Muñoz‐Espín trusts endorse each paper. Same corpus, a different order for every researcher.
- Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
- Senescence surveillance of pre-malignant hepatocytes limits liver cancer development (Nature)
- A complex secretory program orchestrated by the inflammasome controls paracrine senescence (Nature Cell Biology)
- Senescence-messaging secretome: SMS-ing cellular stress (Nature Reviews. Cancer)
- An Oncogene-Induced DNA Damage Model for Cancer Development (Science)
- Senescence is a developmental mechanism that contributes to embryonic growth and patterning. (Cell)
- Blinded by the Light: The Growing Complexity of p53. (Cell)
- Integration of Smad and forkhead pathways in the control of neuroepithelial and glioblastoma cell proliferation. (Cell)
- The forkhead transcription factor Foxo1 regulates adipocyte differentiation. (Developmental Cell)
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
Their papers
- Cellular senescence: from physiology to pathology (2014) — 2,510 citations
- Programmed cell senescence during mammalian embryonic development. (2013) — 1,299 citations