Cheng-Jun Hu

Molecular Biology · 2003–2007

A researcher in the Science corpus: 5 papers; trusts 4 researchers and is trusted by 8 researchers here, as inferred from the citation graph.

Subfields

Whom Cheng-Jun Hu trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Roland H. Wenger
  2. K. Kondo
  3. Liyi Wang
  4. G. Semenza
  5. B. Keith
  6. W. Kaelin
  7. T. Cramer
  8. M. C. Simon

Who trusts Cheng-Jun Hu overall

  1. John D. Gordan
  2. G. Semenza
  3. M. C. Simon
  4. W. Kaelin
  5. Erinn B. Rankin
  6. Jessica A. Bertout
  7. N. Prabhakar
  8. R. Guzy
  9. Huafeng Zhang
  10. C. Wigerup

Papers from Cheng-Jun Hu's perspective

Ordered by how strongly the researchers Cheng-Jun Hu trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Transcription Factor HIF-1 Is a Necessary Mediator of the Pasteur Effect in Mammalian Cells (Molecular and Cellular Biology)
  2. Inhibition of HIF is necessary for tumor suppression by the von Hippel-Lindau protein. (Cancer Cell)
  3. Differential Roles of Hypoxia-Inducible Factor 1α (HIF-1α) and HIF-2α in Hypoxic Gene Regulation (Molecular and Cellular Biology)
  4. FIH-1 is an asparaginyl hydroxylase enzyme that regulates the transcriptional activity of hypoxia-inducible factor. (Genes & Development)
  5. Differentiating the functional role of hypoxia‐inducible factor (HIF)‐1α and HIF‐2α (EPAS‐1) by the use of RNA interference: erythropoietin is a HIF‐2α target gene in Hep3B and Kelly cells
  6. Cellular adaptation to hypoxia: O2‐sensing protein hydroxylases, hypoxia‐inducible transcription factors, and O2‐regulated gene expression (The FASEB Journal)
  7. Hypoxia-inducible Factor-1α Is a Positive Factor in Solid Tumor Growth
  8. Targeting of HIF-α to the von Hippel-Lindau Ubiquitylation Complex by O2-Regulated Prolyl Hydroxylation (Science)
  9. Core transcriptional regulatory circuitry in human embryonic stem cells. (Cell)
  10. Inhibition of HIF2α Is Sufficient to Suppress pVHL-Defective Tumor Growth (PLoS Biology)

Their papers