R. Guzy

Molecular Biology · 2005–2019

A researcher in the Science corpus: 4 papers; trusts 6 researchers and is trusted by 9 researchers here, as inferred from the citation graph.

Subfields

Whom R. Guzy trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Kyle D Mansfield
  2. P. Schumacker
  3. J. Brunelle
  4. G. Semenza
  5. J. Turrens
  6. Roland H. Wenger
  7. Cheng-Jun Hu
  8. B. Hoyos
  9. V. Thannickal
  10. M. C. Simon

Who trusts R. Guzy overall

  1. G. Semenza
  2. V. Gogvadze
  3. P. Schumacker
  4. Michael Ristow
  5. Jung-whan Kim
  6. M. Murphy
  7. J. Cubillos-Ruiz
  8. S. Orrenius
  9. Brian B. Ratliff
  10. S. Cadenas

Papers from R. Guzy's perspective

Ordered by how strongly the researchers R. Guzy trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Faculty Opinions recommendation of Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation.
  2. Characterization of the Human Prolyl 4-Hydroxylases That Modify the Hypoxia-inducible Factor* (Journal of Biological Chemistry)
  3. C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation. (Cell)
  4. Targeting of HIF-α to the von Hippel-Lindau Ubiquitylation Complex by O2-Regulated Prolyl Hydroxylation (Science)
  5. Faculty Opinions recommendation of HIFalpha targeted for VHL-mediated destruction by proline hydroxylation: implications for O2 sensing.
  6. Oxygen sensing requires mitochondrial ROS but not oxidative phosphorylation. (Cell Metabolism)
  7. Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-alpha activation. (Cell Metabolism)
  8. Complex III Releases Superoxide to Both Sides of the Inner Mitochondrial Membrane* (Journal of Biological Chemistry)
  9. A Conserved Family of Prolyl-4-Hydroxylases That Modify HIF (Science)
  10. Cellular adaptation to hypoxia: O2‐sensing protein hydroxylases, hypoxia‐inducible transcription factors, and O2‐regulated gene expression (The FASEB Journal)

Their papers