B. Keith

Molecular Biology · 2003–2016

A researcher in the Science corpus: 7 papers; trusts 7 researchers and is trusted by 3 researchers here, as inferred from the citation graph.

Subfields

Whom B. Keith trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. G. Semenza
  2. W. Kaelin
  3. Cheng-Jun Hu
  4. K. Kondo
  5. M. C. Simon
  6. Roland H. Wenger

Who trusts B. Keith overall

  1. Rakesh K. Jain
  2. John D. Gordan
  3. Cheng-Jun Hu
  4. G. Semenza
  5. M. Laplante
  6. C. Wigerup
  7. M. C. Simon
  8. A. Palazón

Papers from B. Keith's perspective

Ordered by how strongly the researchers B. Keith trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Transcription Factor HIF-1 Is a Necessary Mediator of the Pasteur Effect in Mammalian Cells (Molecular and Cellular Biology)
  2. FIH-1 is an asparaginyl hydroxylase enzyme that regulates the transcriptional activity of hypoxia-inducible factor. (Genes & Development)
  3. Core transcriptional regulatory circuitry in human embryonic stem cells. (Cell)
  4. TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
  5. Hypoxia requires notch signaling to maintain the undifferentiated cell state. (Developmental Cell)
  6. HIF-2alpha regulates Oct-4: effects of hypoxia on stem cell function, embryonic development, and tumor growth. (Genes & Development)
  7. TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
  8. Modulation of hypoxia-inducible factor 1alpha expression by the epidermal growth factor/phosphatidylinositol 3-kinase/PTEN/AKT/FRAP pathway in human prostate cancer cells: implications for tumor… (Cancer Research)
  9. Low O2 tensions and the prevention of differentiation of hES cells. (Proceedings of the National Academy of Sciences of the United States of America)
  10. Regulation of Hypoxia-Inducible Factor 1α Expression and Function by the Mammalian Target of Rapamycin (Molecular and Cellular Biology)

Their papers