Interleukin-17 family members and inflammation.

By J. Kolls, A. Lindén. Immunity, 2004

2,602 citations recorded by the source corpus.

Subfields: Dermatology, Immunology, Pathology and Forensic Medicine

View Paper: https://www.semanticscholar.org/paper/82f7b37e37c60ab7b77ddb536bb7812f8f1291aa

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Dermatology

1. D. Cua
2. C. Weaver
3. Y. Iwakura
4. H. Ishigame
5. T. Korn
6. Paul R. Mangan
7. C. Dong
8. E. Bettelli

In Immunology

1. D. Cua
2. Shizuo Akira
3. C. Dong
4. V. Kuchroo
5. Satoshi Uematsu
6. S. Gaffen
7. A. Jetten
8. R. Medzhitov

In Pathology and Forensic Medicine

1. D. Cua
2. Y. Iwakura
3. C. Weaver
4. V. Kuchroo
5. C. Tato
6. S. Gaffen
7. H. Ishigame
8. L. Harrington

Citations in the corpus

• Anti-interleukin-17 monoclonal antibody ixekizumab in chronic plaque psoriasis. (2012, neutral): “Nasopharyngitis 5 (19) 3 (11) 3 (10) 3 (10) 4 (14)”
• Exploring the full spectrum of macrophage activation (2008, background): “These cells produce IL-17 , a cytokine that is associated with high levels of polymorphonuclear leukocyte (PMN) recruitment to tissues, which can contribute to inflammatory autoimmune pathologies (reviewed in Ref”
• Regulation of humoral and cellular gut immunity by lamina propria dendritic cells expressing Toll-like receptor 5 (2008, background): “As IL-17 can influence cytokine production by a wide range of cell types and can induce the activation and migration of neutrophil”
• Innate IL-17-producing cells: the sentinels of the immune system (2010, background): “A key feature of neutrophil recruitment is the requirement for an early response — within 4–8 hours after exposure — for effective immunity against bacterial and fungal pathogens such as Citrobacter rodentium, Klebsiella pneumoniae, Staphylococcus aureus and Candida albicans”
• IL‐23 and the Th17 pathway promote inflammation and impair antifungal immune resistance (2007, methodology): “As IFN-c is one potent inducer of IDO on PMN [24], we measured the effect of IL-23 or IL-17 on the IFN-c-mediated IDO activation on PMN as well as on matrix metalloproteinase 9 (MMP9)/myeloperoxidase (MPO) production, both linked to IL-17 activation [ 25 ], in”
• IL-17 Production Is Dominated by γδ T Cells rather than CD4 T Cells during Mycobacterium tuberculosis Infection1 (2006, background): “IL-17 induces chemokines, growth factors, and adhesion molecules, and augments neutrophil accumulation (6, 7).”
• Interleukin-23 drives innate and T cell–mediated intestinal inflammation (2006, background): “The IL-17R is widely expressed, and IL-17 binding promotes stromal, endothelial, and epithelial cells to secrete proinfl ammatory mediators that recruit neutrophils to sites of infl ammation (41).”
• IL-17 and Th17 Cells. (2009, background): “Depending on the cellular source of IL-6, cytokines such as IL-1, tumor necrosis factor (TNF), plateletderived growth factor (PDGF), IL-3, GMCSF, and IL-17 are important inducers of IL-6 (39, 40).”